Past the weak association epidemiology, what would the biological mechanism be?
6 thoughts on “Claim: Higher mercury levels in humans associated with increased risk of Type 2 diabetes”
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Past the weak association epidemiology, what would the biological mechanism be?
Comments are closed.
Tee hee hee hee hee — “multiple birthday syndrome” — new to me!
They got the effect from monovalent Hg in 0.5M acetic acid. Not exactly in vitro conditions. Are we seeing mercury insertion in protein disulfides as a part of ingestion of oganomecurials?
A lot of indefinites in the press release. Maybe happens to older people who are “wealthy” enough to live and eat well and who may be overweight. When you get to 65 you are falling apart for all kinds of reasons. Maybe mercury or just maybe the effects of multiple birthday syndrome.
I was waiting for a reference to coal fueled utilities.
Mercury can cause multiple effects because it knocks out selenoenzymes. It can also reduce disulphide bonds, potentially disabling all proteins, including insulin:
http://pubs.acs.org/doi/abs/10.1021/bi00707a002
The question is, do you really get diabetes as a result of mercury poisoning, or do you die from something else first?
Install more flourescent bulbs!!!
I came in contact with mercury in differential flowmeters pressure monitors etc. I have type 2 diabetes,first in my family. coincidence?
I dunno that the weak epidemiology supports the implication and I dunno what the mechanism would be. I do know that, in 1920, the question of whether radiated water was causing disease would have led to the same questions.